Critically assess the changing relationships between TNCs and nation Essay - 1

Critically assess the changing relationships between TNCs and nation states, comparing their respective roles, objectives, flexibility and power - Essay Example In a recent attempt, globalization has been described as â€Å"a process that encompasses the causes, course, and consequences of transnational and trans-cultural integration of human and non-human activities† (Al-Rodhan and Stoudmann, 2006). Information technology (IT) has played a very important role in the globalization of economies across the world. Improvements in information technology from the early 90’s and the growth of disruptive internet technology, computer hardware and software have made it easier for both business enterprises and common people to access information. It has turned the world into a small global village. Improvements in technology have brought down the cost of doing business by all sectors of the economy and contributed in their efficiency gain. Information technology has made it easier for companies to restructure their business practices by modifying their inventory management system and â€Å"just-in-time† production technologies. Nation states have been described as a state which has the basic function of creating a cultural self-identity based on the strength of its national identity. It has been argued that the very creation of the nation state is a type of an invention that signifies a combination of nation and a state. A nation state is connected in terms of its nationality, social and cultural aspects and a strongly organized political system that works in best interest of the nation. The role of the nation state has changed considerably in the era of globalization. In the modern era, the role of the nation state has changed from that of a chief political organization. In the present times they are adapting themselves to become the singular units that can meet the needs of its local people at a time when world economies are integrating strongly. In a research that has been conducted by Zhou (2011) it has been highlighted that sovereignty of nation state has become more complex, conditional and

Using current and historical examples discuss the constraints and Essay

Using current and historical examples discuss the constraints and opportunities for alternative forms of work organisation in the globalized world - Essay Example w decades have given prominence to the understanding of work organization in reference to the advent of globalization; international trade being coupled with an increase in mobility of people and capital. The confusion around globalization makes it harder to concretely define the term itself. In many cases, it is the ‘prominent catchphrase for describing the process of international economic integration’ (Scholte, 2005, 16). In the context of the argument presented in this paper, globalization is viewed as an amalgamation of liberalization, universalization and westernization. Hence, it could be defined as the process of removing restrictions on movements between countries, creating a synthesis of cultures and spreading experiences to people in all corners of the world (Scholte, 2005). Work organisation is often linked with capitalism; an economic system which advocates the prominence of private ownership, wage labour and emergence of market forces as the dictating power in setting prices. Karl Marx referred to capitalist societies as an advanced form of social organization that would pave way for the working class to come to power (Whitley, 2000). The ideology revolved around the transformation of human society being a series of evolution that was controlled by the changing dynamics of the world and the requirements of the general population. Where socialist or communist systems once wielded a stronger control over the processes governing work organization, this was replaced by the capitalistic approach. In the latter approach, the barter system of goods exchange was replaced by introducing an item of use-value; initially gold and then paper money. Marx professed that the next change would be the working class coming to power, with its primary driving factor bein g the exploitation of labour or workforce, under which capitalists try to extract value for the owner or the â€Å"bourgeois† society (Whitley, 2000). Further progression of Marx’s work at the turn

Origins of Tissues Macrophages

Origins of Tissues Macrophages Ryan Lewis    Discuss current ideas about the origins of tissue macrophages and whether these origins influence the subsequent functions of macrophage Graphical Abstract Figure Legend: This figure highlights possible tissue macrophage origins and their development pathways. The essay discusses contradicting findings in the literature, involving three different publications; Sheng et al, 2015 (blue pathway), Hoeffel et al, 2015 (red pathway) and Perdiguero et al, 2015 (green pathway) which are shown in the figure. Cell positions relate to the time point they are established during embryonic development. Positions of yolk sac and foetal liver are also related to the times they are developed. Introduction In the late 19th century Ilya Metchnikoff discovered macrophages (Tauber, 2003) and since then our understanding of the immune system and its complexity has progressed to a stage where the macrophage is no longer as simple as was originally depicted by Metchnikoff. Although much more is known about tissue specific macrophages and their functions, the origins of these macrophages are less well understood including how their origins relate to the functions they have within specific tissues. This essay aims to address the current ideas about the origins of tissue macrophages and whether these origins influence the subsequent functions of macrophages. Macrophage Discovery and History As previously mentioned Metchnikoff discovered the macrophage late in the 19th century (Tauber, 2003). Metchnikoff published a paper talking about phagocytic cells he had observed in frogs, he described the phagocytic cells as being involved in host defence but also the clearing of dead and dying cells (Gordon, 2007). Mechnikoff then discovered the presence macrophages in starfish, which dont have a vascular system, which led him to the discovery of tissue-resident macrophages (Gordon, 2007). Metchnikoff received the Nobel prize for his studies on cellular immunity to infection in vertebrates which he shared with Paul Ehrlich who discovered humoral immunity (Gordon, 2007). It took roughly 80 years after Metchnikoffs discovery before the origin of the tissue macrophage was uncovered. It was proposed that tissue macrophages originated from circulating monocytes in the blood (van Furth and Cohn, 1968), this theory has persisted for the last 40 years however from recent studies we know t hat this is not the primary origin of the tissue macrophage. Shortly after the theory that tissue macrophages originated from circulating monocytes was proposed, it was discovered that tissue macrophages and monocytes are heterogenous and their heterogeneity is conserved in humans and mice (Gordon and Taylor, 2005). The discovery of monocyte subsets followed shortly after in 1983, which supported the theory that tissue macrophages originated from circulating monocytes (Yona and Jung, 2009). The theory that tissue macrophages are derived from circulating monocytes has been the prevailing view until very recently partly due to the arrival of advanced techniques including; fate mapping and ionizing radiation. In the last 5-6 years, many definitive publications have redefined our understanding of the origins of tissue macrophages (Epelman et al, 2014). Recent studies have shown that many tissue macrophages are established during embryonic development and continually self-replenish into adulthood independently of any input from circulating monocytes in the blood (Epelman et al, 2014; Ginhoux et al, 2010; Hashimoto et al, 2013; Yona et al, 2013). Tissue Macrophage Heterogeneity and Function Tissue macrophage have a huge degree of heterogeneity which reflects upon the specialization of their functions in different tissues and locations (Gordon and Taylor, 2005). Macrophage heterogeneity is required to ensure the tissue macrophage has the most effective phenotype to tackle its specific microenvironment, this is particularly important in the gut. Tissue macrophages in the gut isolated from the lamina propria have a unique phenotype characterised by high phagocytic and bactericidal activity but very poor production of pro-inflammatory cytokines which makes them perfectly suited to their microenvironment (Gordon and Taylor, 2005). There are many specialised tissue macrophages that have very distinct functions including; osteoclasts in the bone which breakdown bone deposits for bone remodelling, alveolar macrophages (dust cells) in the lung that break down foreign material and pathogens, and microglia in the brain which play a role in neuronal development homeostasis and the recovery from pathology (Boyce et al, 2008; Rubins, 2003; Prinz et al, 2014). The theory that tissue macrophage populations are replenished from circulating monocytes in the blood is somewhat true but the most diverse tissue macrophages such as microglia, alveolar macrophages and osteoclasts are replenished through self-renewal and proliferation (Yona and Jung, 2009). There is a substantial number of studies discussing whether macrophages originating from monocytes in the blood can differentiate into resident tissue macrophages. In most cases the monocyte subset that the macrophage originated from determines its ability to differentiate into a specialized resident tissue macrophage, this is particularly true in the lung as studies have shown only Ly6Clo, not Ly6Chi, monocytes have the ability to differentiate into enchymal lung macrophages (Landsman et al, 2007). In regards to the more complex and specialised alveolar macrophages in the lung, studies have shown that these macrophage s require a parenchymal lung macrophage intermediate (Landsman and Jung, 2007). Circulating monocytes in the blood were long believed to be the origin of specialised tissue macrophages but recent evidence has shown that this is incorrect and proven that many of these tissue macrophage populations are developed long before birth (Epelman et al, 2014). Origins of Tissue Macrophages Macrophages are first observed during embryonic day 6.5 and are produced in the yolk sac during what is termed as primitive haematopoiesis (Epelman et al, 2014). During this early stage in development macrophages are the only immune cell produced due to restricted progenitors in the yolk sac. During embryonic days 8.5 10.5 hematopoietic stem cells (HSCs) emerge from the aorta-gonad meso-nephros (AGM) and give rise to all immune lineages (Epelman et al, 2014). At embryonic day 10.5 HSCs migrate from the AGM to the foetal liver, the foetal liver then becomes the major hematopoietic organ until birth. Only after birth do bone marrow HSCs become the primary progenitors and produce all immune lineages (Orkin and Zon, 2008). Microglia are the only tissue macrophages that are established in the yolk sac and are self-maintained through-out adulthood, all the other tissue macrophages are established from embryonic day 14.5 to birth and either self-maintained by proliferation or replenished b y HSCs in the bone marrow (Ginhoux et al, 2010; Sheng et al, 2015). The arrival of fat-mapping techniques have enabled researchers to precisely track embryonic macrophage populations into adulthood, giving an insight into the relationship between resident tissue macrophages and circulating blood monocytes (Epelman et al, 2014). As previously discussed, microglia are the only tissue macrophage originating from the yolk sac and arise before embryonic day 8 (Ginhoux et al 2010). Fate mapping analysis was used to determine that the origin of microglia was the primitive myeloid precursors in the yolk sac and also proved that microglia are self-maintained independently of any circulating blood monocytes (Ginhoux et al, 2010). There is also evidence that Langerhans cells originate from the yolk sac but only partially (Sheng et al, 2015). The fate mapping study by Sheng proved that microglia and Langerhans cells were the only tissue macrophages that originate from yolk sac precursors and th at most adult tissue macrophages originate from a second wave of haematopoiesis driven by HSCs. (Sheng et al, 2015). The number recent of publications concerning tissue macrophage origins is staggering and is most likely attributed to the arrival of fate mapping techniques. With the large surge of new studies regarding tissue macrophage origins it is important that a clear understanding is generated but this is not always possible with such a complicated subject. Contrasting Studies into Tissue Macrophage Origins There are a few recent studies concerning tissue macrophage origins which are particularly interesting. Sheng (Sheng et al, 2015) arrived at the conclusion that most tissue macrophages originate from HSCs however there are a few publications which contradict Shengs findings. Perdiguero concluded that yolk sac derived erythro-myeloid progenitors, were origin of almost all tissue macrophages which contrasts greatly with Shengs observations. (Perdiguero et al, 2015). Perdiguero also concluded that microglia were derived from erythro-myeloid progenitors rather than primitive yolk sac progenitors that was observed by Sheng, although both do come from the yolk sac (Perdiguero et al, 2015; Sheng et al, 2015). Perdiguero predicted that almost all other tissue macrophages originated from erythro-myeloid progenitors (Perdiguero et al, 2015; Sheng et al, 2015). A study by Hoeffel aligned well with Perdigueros observations but Hoeffel observed that primitive yolk sac progenitors gave rise to mic roglia rather than erythro-myeloid progenitors that was observed by Perdiguero (Hoeffel et al, 2015; Perdiguero et al, 2015). As well as the difference in the development of microglia, Hoeffel predicted that erythro-myeloid progenitors migrated to the foetal liver, giving rise to foetal monocytes which were then responsible for the production of tissue macrophages. (Hoeffel et al, 2015). Each of these 3 examples also propose a separate proposed major path of ontogeny and differentiation to adult tissue macrophage state. Perdiguero proposes erythro-myeloid progenitors from the yolk sac as the major precursor of tissue macrophages, Heoffel proposes erythro-myeloid progenitors from the foetal liver, as foetal monocytes, as the major precursor and, Sheng proposes that HSCs from the foetal liver are the major precursor (Perdiguero et al, 2015; Hoeffel et al, 2015; Sheng et al, 2015; Guinhoux and Guilliams, 2016). Although the observations made by Sheng are profoundly different to those m ade by Perdiguero and Hoeffel it could be down to the fate mapping technique they used. The model they used is not adapted to distinguish between late erythro-myeloid progenitors and foetal HSCs which has clearly effected the conclusion they have come to (Guinhoux and Guilliams, 2016). Although fate mapping has great potential in advancing our knowledge of cellular ontogeny there are certain limitation that come with it and these limitations must be considered when designing experiments and analysing data (Guinhoux and Guilliams, 2016). Do Tissue Macrophage Origins Matter? Determining the origins of tissue macrophages may be valuable for furthering our knowledge and understanding of their development but do their origins have any influence in determining their function? As well as ontogeny, diversity in the functions of tissue macrophages can also be attributed to the local signals received by the macrophages. These local changes can drive the expression of unique transcription factors which in turn lead to different functions (Lavin et al, 2015). There is a lot of evidence to suggest that the tissue macrophages microenvironment can alter its function, the plasticity of tissue macrophages allows them to adjust their functions to inflammatory events (Lavin et al, 2015). Using ionizing radiation most embryonic-derived tissue macrophages can be eliminated, they can then be replaced with donor-derived bone marrow progenitors to determine if the wild type state of the tissue can be restored. Using this technique, studies have proven that bone marrow progeni tors can completely restore the enhancer profile and transcriptional programme of the embryonic-derived tissue macrophages that were eliminated (Lavin et al, 2015). A very recent study has shown that yolk sac macrophages, foetal liver monocytes and adult bone marrow monocytes can all successfully differentiate into alveolar macrophages in the lung after the removal of the native alveolar macrophages using ionizing radiation (van de Laar et al, 2016). The study also showed that other already developed tissue macrophages, liver, peritoneal and colon macrophages cannot successfully differentiate into alveolar macrophages in the lung. This finding suggests that the plasticity of the mononuclear phagocyte system is at its largest during the precursor stage and after differentiation to tissue-resident macrophages no further phenotypic changes of macrophage types can take place (van de Laar, 2016). Perhaps the most interesting finding from this study is that the alveolar macrophages differ entiated from yolk sac macrophages, foetal liver monocytes and bone marrow monocytes were still able to self-maintain and prevent alveolar proteinosis (van de Laar, 2016). Similar results have also been observed with Kupffer cells. Kupffer cells were eliminated from the liver using diphtheria toxin-mediated depletion allowing its niche to become vacant. Observations showed that circulating monocytes can engraft the liver and adopt the transcriptional profile of the eliminated Kupffer cells and also become long-living self-renewing cells like their eliminated counterparts (Scott et al, 2015). These new findings question whether the origin of tissue macrophages is truly important to their function as the progenitors and monocytes tested have all been able to restore the tissues lost macrophages successfully without any loss of function. Conclusion Although determining the origins of tissue macrophages and other members of the immune system is important for the progression of our knowledge it remains to be seen whether the actual origins have any implications on the function of the tissue macrophages. The techniques used in the publications discussed are still very new and still require refinement, I believe further refinement of the techniques will enable a more detailed and accurate description on the origins of tissue macrophages and the role the origins play in their function. References       BOYCE, B.F., YAO, Z. XING, L. 2009Osteoclasts have multiple roles in bone addition to bone resorption.Critical Reviews in Eukaryotic Gene Expression, 19.3, 171-180 EPELMAN, S., LAVINE, K.J. RANDOLPH, G.J. 2014Origin and functions of tissue macrophages.Immunity, 41.1, 21-35 GINHOUX, F., GRETER, M., LEBOEUF, M., NANDI, S., SEE, P., GOKHAN, S., MEHLER, M.F., CONWAY, S.J., GUAN NG, L., STANLEY, E.R., SAMOKHVALOV, I.M. MERAD, M. 2010Fate mapping analysis reveals that adult microglia derive from primitive macrophages.Science, 330.6005, 841-845 GUINHOUX, F. GUILLIAMS 2016Tissue-resident macrophage ontogeny and homeostasis.Immunity, 44.3, 439-449 GORDON, S. 2007The macrophage: past, present and future.European Journal of Immunology, 37, 9-17 GORDON, S. TAYLOR, P.R. 2005Monocyte and macrophage heterogeneity.Nature Reviews: Immunology, 5.12, 953-964 HASHIMOTO, D., CHOW, A., NOIZAT, C., TEO, P., BEASLEY, M.B., LEBOEUF, M., BECKER, C.D., SEE, P., PRICE, J., LUCAS, D., GRETER, M., MORTHA, A., BOYER, S.W., FORSBERG, E.C., TANAKA, M., VAN ROOIJEN, N., GARCIA-SASTRE, A., STANLEY, E.R., GINHOUX, F., FRENETTE, P.S. MERAD, M. 2013Tissue-resident macrophages self-maintain locally throughout adult life with minimal contribution from circulating monocytes.Immunity, 38.4, 792-804 HOEFFEL, G., CHEN, J., LAVIN, Y., LOW, D., ALMEIDA, F.F., SEE, P., BEAUDIN, A.E., LUM, J., LOW, I., FORSBERG, E.C, POIDINGER, M., ZOLEZZI, F., LARBI, A., NG, L.G., CHAN, J.K., GRETER, J.K., BECHER, B., SAMOKHVALOV, I.M., MERAD, M. GINHOUX, F. 2015C-Myb(+) erythro-myeloid progenitor-derived fetal monocytes give rise to adult tissue-resident macrophages.Immunity, 42.4, 665-678 LANDSMAN, L., VAROL, C. JUNG, S. 2007Distinct differentiation potential of blood monocyte subsets in the lung. Journal of Immunology, 178.4, 2000-2007 LANDSMAN, L. JUNG, S. 2007Lung macrophages serve as obligatory intermediate between blood monocytes and alveolar macrophages.Journal of Immunology, 179.6, 3488-3494 LAVIN, Y., MORTHA, A., RAHMAN, A. MERAD, M. 2016Regulation of macrophage development and function in peripheral tissues.Nature Reviews: Immunology, 15.12, 731-744 ORKIN, S.H. ZON, L.I. 2008Haematopoiesis: an evolving paradigm for stem cell biology.Cell, 132, 631-644 PERDIGUERO, E.G., KLAPPROTH, K., SCHULZ, C., BUSCH, K., AZZONI, E., CROZET, L., GARNER, H., TROUILLET, C., DE BRUIJN, M.F., GEISSMANN, F. RODEWALD, H.R. 2014Tissue-resident macrophages originate from yolk-sac-derived erythro-myeloid progenitors.Nature, 518, 547-551 PRINZ, M., TAY, T.L., WOLF, Y. JUNG, S. 2014Microglia: unique and common features with other tissue macrophages.Acta Neuropathologica, 128.3, 319-331 RUBINS, J.B. 2003Alveolar macrophages: wielding the double-edged sword of inflammation.American Journal of Respiratory and Critical Care Medicine, 167.2, 103-104 SCOTT, C.L., ZHENG, F., DE BAETSELIER, P., MARTENS, L., SAEYS, Y., DE PRIJCK, S., LIPPENS, S., ABELS, C., SCHOONOOGHE, S., RAES, G., DEVOOGDT, N., LAMBRECHT, B.N., BESCHIN, A. GUILLIAMS, M. 2016Bone marrow-derived monocytes give rise to self-renewing and fully differentiated Kupffer cells.Nature Communications, 7, 10321 SHENG, J., RUEDL, C. KARJALAINEN, K. 2015Most tissue-resident macrophages except microglia are derived from fetal hematopoietic stem cells.Immunity, 43.2, 382-393 TAUBER, A.I. 2003Metchnikoff and the phagocytosis theory.Nature Reviews: Molecular Cell Biology, 4, 897-901 VAN DE LAAR, L., SAELENS, W., DE PRIJCK, S., MARTENS, L., SCOTT, C.L., VAN ISTERDAEL, G., HOFFMANN, E., BEYAERT, R., SAEYS, Y., LAMBRECHT, B.N. GUILLIAMS, M. 2016Yolk sac macrophages, fetal liver, and adult monocytes can colonize an empty niche and develop into functional tissue-resident macrophages.Immunity, 44.4, 755-768 VAN FURTH, R. COHN, Z.A. 1968The origin and kinetics of mononuclear phagocytes.The Journal of Experimental Medicine, 128.3, 415-435 YONA, S., KIM, K.W., WOLF, Y., MILDNER, A., VAROL, D., BRECKER, M., STRAUSS-AYALI, D., VIUKOV, S., GUILLIAMS, M., MISHARIN, A., HUME, D.A., PERLMAN, H., MALISSEN, B., ZELZER, E. 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Health Effects of High Fructose Corn Syrup :: Sugar HFCS Artificial Sweetener Food

Health Effects of High Fructose Corn Syrup Abstract: What is high fructose corn syrup? Is it some kind of disease making machine? High fructose corn syrup is causing many problems in the United States and two of the main problems are obesity and type two diabetes. There are also many other diseases that branch off from these two main diseases, like liver and heart disease. In this paper, the reason why high fructose corn syrup is becoming such a huge problem in the United States will be explored. The Japanese first developed high fructose corn syrup. When they saw that it is more cost effective than other sugars, other companies started to use high fructose corn syrup in their products. High fructose corn syrup is a sugar made of fifty-five percent fructose and forty-five percent glucose. Compared to normal table sugar, which is fifty percent fructose and fifty percent glucose, it doesn?t seem like much. Yet the fructose in high fructose corn syrup is less attached from normal sugar. This fructose that is free from the glucose part is more harmful to the body. It was first introduced in 1970 but it hasn?t been actively used until the late 1900?s. Yet because we started to use high fructose corn syrup, it is now causing a lot of problems. It has many negative side effects after consumption, and most of the consuming is done in the United States. Two of the main problems caused by high fructose corn syrup are obesity and type two diabetes. Some of the other branches of these problems are liver and heart disease, osteoporosis, an increase in triglycerides, and many other kinds of health problems. Since high fructose corn syrup is very common in our daily diets, ranging for drinks to desserts, it is becoming a huge problem. (Severson, K. 2004) One of the main problems of high fructose corn syrup is obesity. Eating high fructose corn syrup is like eating fat. Fructose isn?t absorbed the large intestine or the first part of the small intestine. Fructose is absorbed in the jejunum at a much higher rate than normal glucose. After it is absorbed, it goes to the liver, where it is converted to fatty acids. When someone consumes too much fructose, the liver is unable to convert all of it so it can be absorbed improperly.

“White Man’s Burden” and “Shooting an Elephant”

In the poem â€Å"White Man’s Burden† and essay â€Å"Shooting an Elephant† is talk about the two white men has a different point of views about imperialism and how the white treat the natives. â€Å"White Man’s Burden† by Rudyard Kipling is talk about how does white man sacrifice for the native during the imperialism. â€Å"Shooting an elephant† by George Orwell is talk about how does the white treat the natives by reflect the way shooting an elephant. â€Å"White Man’s Burden† by Rudyard Kipling, the first identity of the white man is sacrifice for the natives, he was one believed in the virtues of imperialism in that period. As the text proof â€Å"send forth the best ye breed-go bind your sons to exile, to serve your captives need; to wait in heavy harness†. The white man try to make difference to the natives, educated them was white man’s job and the cast it as their goal. The second identity of the white man is racial and cultural stereotype, as the text proof â€Å"Ye dare not stoop to less-nor call too loud on freedom to cloak your weariness, by all ye cry or whisper, by all ye leave or do. The white man have to be open mind about different culture and to think about the way of white man teaching the native, be open mind to accept the natives culture as well too. â€Å"Shooting an Elephant† by George Orwell, Through the essay Orwell reflects the social condition at Burma as a result of British Imperialism and his own view on imperialism. The first identity of the white police officer was sub-divisional police officer of the town, an in an aimless. Second identity of the white p olice officer as he expresses is great sympathy for the oppressed Burmese. White police officer reflects the way he Killed the elephant, emotionally. As the text proof â€Å"as soon as I saw the elephant I knew with perfect certainty that I ought not to shoot him. I did not in the least want to shoot him, I decide I could watch him a little bit make sure he did not turn savage again, and then go home. With the magical rifle in my hands I was momentarily worth watching. Suddenly I realized that I should have shot the elephant after all†¦Ã¢â‚¬ ¦Ã¢â‚¬  and finally he shot the elephant. In the essay the police officer feels truly stuck between his loyalty to Britain and his sincere distaste of imperialism. The police officer was kind open mind about the time of imperialism. Ironically â€Å"he shoot the elephant† in order to maintain the integrity o the system of imperialism, In Orwell opinion imperialism take away peoples think, doing whatever to the expectation of other. â€Å"The White man’s burden† compare to the â€Å"Shooting an elephant† they are both white, and the winner of the imperialism. But they have different view and thought about the imperialism. In the poem â€Å"The white man’s burden† the white man to bring civilization to the natives, by educate them and take responsible to the native, and white man sacrifice a lot to the natives . In the essay â€Å"shooting an elephant† the white police officer think that the imperialism was an evil thing to the natives, his sympathy for the oppressed native, but in other hand he can’t do anything about it. In conclusion the poem and essay was written during the Imperialism period, causing many mix feelings between what is good and what is bad? But I feel so bad about the natives, why should white people have to care about native? Because if you want to control over a country you need to know their culture and respect their culture this is call winner. Why is important? Because we are human and we have to try everyone equally the way they should be treat. In shooting an elephant, the white police know that imperialism is evil thing to the native, at the time he couldn’t do anything about. But today is different and we all equal respect each other, and live in freedom country.

Promote Creativity And Creative Learning Essay

Creative learning is all about helping children develop their imaginative skills through exploration of different materials and ways of expressing themselves, for example this can be methods like dance, ICT, building and also traditional creative methods like painting and drawing. Creativity itself is all about allowing children to express and explore themselves and take risks in doing so. This doesn’t necessarily have to be in a defined method but could simply be their own play, for example their role play is an expression of their creativity. Most theories about young children view children as highly creative which allows them to explore and experiment with the world around them. Creativity is more about the process than it is about the actual end product as it allows us to learn more about ourselves, like what we’re good and not good at. ‘Creative Partnerships’ was a scheme set up with the term ‘creative learning’ to sum up their programme. They believed working together to try and create new approaches to learning would stimulate the people learning by giving them a new approach and bring the curriculum to life. Another approach the ‘creativity, find it and promote it’ challenged practitioners to be more critical towards the way they think about creativity. It challenged them to try and create connections they wouldn’t normally make and constantly question conventions that they normally wouldn’t. Creativity and creative learning can be beneficial for children for many different reasons. Creativity can give children a way to express and develop their emotions, an example of this may be if a child is sad they may draw a sad picture and then someone has the opportunity to ask them how they feel about it and express themselves. Creativity is also a good way for children to develop socially, an example of this if a child enjoys creative dance then they may wish to create dance routines with other children and this allows them to be both social but develop their communicational and physical development. Creativity can also help children develop intellectually by doing creative problem solving and critical thinking. Children require long unhurried periods of time to develop and express their creativity so they are not rushed into creating something they did not want to. Allowing them as much time as they need, will help them feel like they have created the best thing they could have possibly made as well as allow them to explore original ideas further with the extra time that they have.